![]() Please use the white oil clay for fixing eyes as much as possible to prevent pigmentation in the eyes. There may be slight differences between both eyes since they are produced by handwork. EYESĭefects that are not visible when the eyeball is assembled are not considered as a defective.Īir bubbles and slight scratches may occur due to the process of eyeball production. Resin dolls are gradually discolored over time due to the nature of the material,Įspecially it discolors faster when exposed to direct sunlight or indoor lighting for a long time, so please be careful in storage. In case of the dark resin color, it may be subject to marbling effect and speckles on the doll skin.ĭark fabric may stain doll if worn for a prolonged period of time.Ĭolor fading or bleaching may occur when washing with chemicals(thinner, acetone, etc.). In the process of making doll, Seam, raw material injection hole, small air bubble can be occurred.ĭepending on the production period, color differences may occur even the same color option. 'Removing the seam' is not included to 'Body blushing' option.įrequent movement of a ball jointed doll can rub or damage the ‘Body blushing’. Wavefront analysis of ocular and corneal aberrations is a useful adjunct to corneal topography in the diagnosis of keratoconus and pellucid marginal degeneration.The photos on the product page are sample images so it may vary depending on the setting of the screen.įace-up and body blushing can have differences since they are produced by handwork. A close review of these Zernike terms, in particular, can assist in the differentiation of pathologic steeping of the cornea from otherwise symmetric or regular astigmatism. Conclusions: Keratoconus and pellucid marginal degeneration are characterized by inferior corneal thinning and steeping that induces third order coma–like and trefoil–like aberrations. A review of total eye wavefront data showed that vertical coma is significantly increased in KCN (p=.02) but not PMD, and that vertical trefoil is selectively increased in PMD (p=.03). Analysis of corneal first surface elevation data differentiated keratonic and pellucid eyes from controls, but not from each other, as several cases had a forme fruste presentation. Pellucid eyes were marked by against–the–rule astigmatism, which was not as readily seen in the other groups (p<.01), and an inferiorly displaced thinnest point (p<.01) identified via optical pachymetry (mean –1.5 mm position in the y–axis versus 0.0 control and –0.5 KCN). Keratoconic eyes had thinner corneas on average (mean 461 + 122 µm KCN, 541 + 37 µm control, 493 + 81 µm PMD) than both control and pellucid counterparts (p=.03). Results: Compared to control eyes, keratoconic and pellucid corneas had a significantly wider distribution of corneal power within Orbscan–derived 3 and 5 mm central zones (p<0.01). Some wavefront data was excluded because of poor centroid detail or due to small, undilated pupil size. Hartmann–Shack wavefronts (Zywave, Bausch & Lomb, Rochester, NY) were statistically analyzed at a 5 mm pupil for 6 normal, 19 keratoconic, and 3 pellucid eyes. Slit–scanning videokeratography (Orbscan II, Bausch & Lomb, Salt Lake City, UT) was performed on each eye. Methods: A retrospective review identified patients with a clinical diagnosis of keratoconus (n=49), pellucid marginal degeneration (n=14), and a control group with central corneal power > 47 diopters in at least one meridian (n=11). Purpose: To compare the ocular and corneal wavefront aberrations in control eyes to eyes with a clinical diagnosis of keratoconus (KCN) or pellucid marginal degeneration (PMD).
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